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Home » Hyperplastic Pulpitis

Hyperplastic Pulpitis

July 2, 2025 by Joankessler parkland Leave a Comment

Hyperplastic Pulpitis

Question. What is pulpitis? Explain the different causes of pulpitis.
Describe clinical features and the histopathological picture of chronic open hyperplastic pulpitis.

Answer.

Pulpitis:

Pulpitis refers to the inflammation of the dental pulp within the tooth.

Causes of pulpitis

  • Reversible pulpitis is caused by an agent capable of injuring the pulp, such as trauma, disturbed occlusal relationship, or thermal shock.
  • Irreversible pulpitis: It is caused by the bacterial involvement of pulp through caries, chemical or thermal, or mechanical injury.

“Role of chronic irritation in hyperplastic pulpitis”

Chronic open hyperplastic pulpitis

  • It is also known as chronic hyperplastic pulpitis or pulp polyp, or pulpitis aperta.
  • It is a productive pulpal inflammation due to an extensive carious exposure of the young pulp. It is characterized by the development of granulation tissue covered by epithelium and resulting from long-standing low-grade infection.

pulp polyp treatment

hyperplastic pulpitis

“Clinical examination for hyperplastic pulpitis”

Clinical Features

  • Pulp polyp appears as a small, pinkish, red-lobulated mass, which protrudes from pulp chamber and fills up the carious cavity.
  • The condition is seen in young adults and children. It commonly develops in deciduous molars and first permanent molars.
  • The affected tooth has a large open carious cavity, which has been present for a long duration.
  • The lesion bleeds profusely on provocation.
  • The involved tooth is painless and is sensitive to thermal stimuli.

“Pathophysiology of hyperplastic pulpitis explained”

Histopathology

Diseases of the Pulp and Periapical Tissues Pulp polyp chronic hyperplastic pulpitis

“Root canal therapy for hyperplastic pulpitis”

  • Hyperplastic pulp tissue lesion presents the feature of granulation tissue mass, consisting of numerous proliferating fibroblasts and young blood capillaries.
  • Inflammatory cell infiltration by lymphocytes, plasma cells, and sometimes polymorphonuclear neutrophils in tissue is common.
  • Stratified squamous epithelium is present on the surface of hyperplastic pulpitis, which resembles oral epithelium.
  • The epithelial surface shows well-formed rete peg formation.
  • The epithelial cells on the surface are believed to be desquamated epithelial cells that came either from the buccal mucosa or salivary gland ducts.

Filed Under: Oral Pathology

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